A Methodology for Determining the Mineral Content and Particle Size Distribution of Airborne Coal Mine Dust

"Exposure to airborne dust in coal mines has caused respiratory disease in coal miners. The causal agent in these diseases remains unknown, although the minerals found in and around the coal seam have been implicated. Toxicological studies designed to establish the effect of different mineral combinations and concentrations are lacking because the mineral content of coal mine dust has not been defined. This paper reports the initial results of a study done to characterize the mineral content of the coal mine dust found in a mine in the Upper Freepott seam in Northern West Virginia. Coal mine dust samples were collected using 4- stage cassette impactors at nine locations in the mine. The samples were analyzed by an x-ray microdiffraction film technique supplemented by energy dispersive scanning electron microscopy. Primary size modes of about 17 to 20 micrometers from the samples collected at the haulageway and the returnside of the continuous miner were found. From the returnside of the roof bolter samples, a primary size mode of 12to15 micrometers was measured. The minerals found in high percentages were illite, kaolinite, quartz, and calcite. The distribution of minerals in inter· and intra- location samples showed wide variations. Trends in these variations are presented. Stobbe, T. J.; Plummer, R. W.; Kim, H.; Jones. W.: Characterization of coal mine dust. Appl. Ind. Hyg. 2:95· 100; 1986. IntroductionDisease-associated dusts in coal mine environments continue to afflict miners in today's modern mines. The relationship between exposure to coal mine dust and the development of various respiratory diseases has been well recognized; however, the reasons for variation in occurrence of these diseases among miners in different geographic areas,(1) rank of coal seams,(2-3) and jobs (4) remain uncertain. Several papers have reported on the causal agent in the development of these diseases. Davis (5) found that the percentage of the non-coal minerals present in the lungs of deceased miners increased as the level oi pneumoconiosis became more severe. Reisner (6) reported that cell damage caused by coal mine dust increases with the mineral and quartz content of the dust and with the geo· logical age and rank of the coals. Two studies, investigating the in vitro effects of mineral dusts. reported by Seemayer and Manojlovic (7) also obtained positive correlation between cytotoxicity and the mineral content of coal mine dust. Gormly (8) postulated that destruction of macrophages by certain mineral dusts is the first step in the development of pneumoconiosis. In 1980, Adamis and Timar (9) reported that quartz. sandstone, slate. respirable colliery dust. bentonite, illite, and some kaolins had cytotoxic effects on rat peritoneal macrophages""in vitro."" Walton,(10) however, reported that the presence of high clay, illite, and kaolin mineral exposures reversed the hazards associated with increasing quartz exposures. This finding was supported by Le Bouffant and coworkers.(11) They reported that illite present in coal mine dust strongly inhibited the pathogenic effects of quartz while kaolin had a weaker inhibitory effect."